Albumin is synthesized by hepatic parenchyma cells at a rate dependent on colloidal osmotic pressure and dietary protein intake.
The rate of albumin synthesis is also subject to feedback regulation determined by the plasma albumin concentration.
Maintenance of plasma albumin concentrations can be achieved with only 10% of normal hepatocyte mass.
The half-life of albumin is 21 days.
Traces of albumin can be found in almost all extracellular body fluids.
Little is lost from the body by excretion.
It is catabolized in various tissues, which are taken up by cells by pinocytosis.
Its constituent amino acids are released by intracellular proteolysis and returned to the body pool.
With any liver disease, there is a decrease in serum albumin, reflecting impaired synthesis.
If liver function is normal and serum albumin is low, this may reflect low protein intake (malnutrition) or protein loss (nephrotic syndrome, malabsorption, or protein-losing enteropathy).
ALBUMIN
Title JUVENILE POLYPS
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